Testosterone enanthate and equipoise cycle

Testosterone is transported in blood bound to a carrier protein, called either testosterone binding globulin (TeBG) or, more commonly, sex hormone binding globulin (SHBG). In healthy men, only about 2% of the circulating testosterone is unbound, with 44% bound to SHBG and 54% bound to serum albumin. The protein-bound testosterone is protected from metabolism in the liver and provides an easily accessible pool of hormone, as the testosterone readily dissociates from its binding protein. Levels of SHBG in plasma are regulated by androgens, oestrogens and thyroid hormones. In healthy men, SHBG levels are fairly constant, but may need to be considered when steroid replacement therapy is used. Plasma testosterone concentrations are around 9–41   nmol/L in healthy men and 1–3   nmol/L in women. Plasma testosterone tends to decrease with age in men. Testosterone is metabolized in the liver, mostly to form androsterone and aetiocholanolone, which are excreted in urine.

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  • Testosterone

There is no set time frame of use for this compound. Regardless of your Testosterone Enanthate doses, this compound can be used for indefinite periods of time. For the TRT patient, it will be used continuously and non-stop as discontinuing use would return the individual to a low testosterone state. For the performance athlete, full cycles will normally last from 8-12 weeks with 16 weeks not being too uncommon. At this stage, most will want to come off and enter into a Post Cycle Therapy (PCT) stage. For the more advance users who live and follow a very hardcore lifestyle, they may simply lower the dose to a TRT level in a bridge phase. However, this type of use presents a lot of risk and cannot be recommended to most. As for stacking, Testosterone Enanthate stacks well with all anabolic steroids.

Core: Active substance: reserpine mg dihydroergocristine mesylate mg (corresponding to mg base dihydroergocristine) clopamide mg Excipients:lactose monohydrate, povidone, stearic acid, talc (magnesium silicate), corn starch; SHELL: iron oxide red (Sikofarm red 30), titanium dioxide, peanut oil, hydrogenated, macrogol 6000, stearic acid, alcohol, cetyl, silica kolloidnsh, bezvvodny, povidone, microcrystalline cellulose, talc (magnesium hydrosilicate) sucrose. Pharmacodynamics Brinerdin is a combination antihypertensive agent that contains in its composition three mutually supportive components. Testosterone enanthate half life – dihydrogenated ergot… Read More Read More

We investigated the effects of long-term testosterone replacement in hypogonadal and elderly men on lipids and lipoproteins. Twenty-two men with initial serum testosterone concentrations below ng/ml took part in the study: 11 with hypopituitarism (1st group) and 11 otherwise healthy elderly men with low testosterone levels (2nd group). Testosterone deficiency was replaced by intramuscular injections of testosterone enanthate 200 mg every second week. Plasma levels of sex hormones, gonadotropins, SHBG, lipids and lipoproteins were determined before the treatment and after 3, 6 and 12 months of treatment. During the treatment serum testosterone and estradiol increased significantly, reaching normal levels. This was associated with a decrease in total cholesterol (from 225 ± mg/dl to 202 ± mg/dl after 6 months and 198 ± mg/dl after 1 year of testosterone administration, P < in men with hypoandrogenism associated with aging and from 255 ± mg/dl to 214 ± mg/dl after 6 months and 206 ± 9 mg/dl after 1 year of treatment, P < in men with hypopituitarism) and LDL-cholesterol concentrations (from 139 ± mg/dl to 126 ± mg/dl after 6 months and 118 ± mg/dl after 1 year of testosterone administration, P < in men with hypoandrogenism associated with aging and from 178 ± mg/dl to 149 ± mg/dl after 6 months and 140 ± mg/dl after 1 year of treatment, P < in men with hypopituitarism). However, no significant decrease in HDL-cholesterol levels or HDL 2 - and HDL 3 -cholesterol subfractions was observed. The effects of testosterone replacement therapy on lipids and lipoproteins were similar in both groups with different aetiology of hypogonadism. No side effects on the prostate were observed. The results of this study indicate that testosterone replacement therapy in hypogonadal and elderly men may have a beneficial effect on lipid metabolism through decreasing total cholesterol and atherogenic fraction of LDL-cholesterol without significant alterations in HDL-cholesterol levels or its subfractions HDL 2 -C and HDL 3 -C.

Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendency to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic steroid on serum lipids is dependant on the dose, route of administration (oral vs. injectable), type of steroid (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Anabolic/androgenic steroids may also adversely effect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardial infarction.

Testosterone enanthate and equipoise cycle

testosterone enanthate and equipoise cycle

Core: Active substance: reserpine mg dihydroergocristine mesylate mg (corresponding to mg base dihydroergocristine) clopamide mg Excipients:lactose monohydrate, povidone, stearic acid, talc (magnesium silicate), corn starch; SHELL: iron oxide red (Sikofarm red 30), titanium dioxide, peanut oil, hydrogenated, macrogol 6000, stearic acid, alcohol, cetyl, silica kolloidnsh, bezvvodny, povidone, microcrystalline cellulose, talc (magnesium hydrosilicate) sucrose. Pharmacodynamics Brinerdin is a combination antihypertensive agent that contains in its composition three mutually supportive components. Testosterone enanthate half life – dihydrogenated ergot… Read More Read More

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