What amazed me about the relationship between Hypothyroidism and testosterone: For decades I had trouble with muscle development. I have been using synthroid for my Hashimotos Hypothyroidism. I recently switch to Naturethroid (a natural bio-identical hormone replacement,) and within 8 weeks, my muscles began to grow! I don’t think that changing from synthetic to natural analog was as much to blame for the growth as was the serendipitous increase in Free T3. I have a conversion problem that kept me from converting the T4 into T3. The Free T3 levels had always been within a “normal” range but at the bottom third of that range. Titrating The Naturethroid, which contains T3 as well as T4, to achieve Free T3 levels in the upper third of the range changed everything. I’ve not checked Free testosterone, but anecdotally — dramatic muscle increases globally, and oddly enough my voice decreased into a deeper tone. Overall more cheerful and clear headed.
So if I understand correctly you are saying that you did an Ostarine cycle, then didn’t do a PCT at all in any capacity, and then after 4 weeks of being off Ostarine you started up another cycle? To me it sounds like your issue is simply deriving from the fact that you started another cycle without getting your estrogen and testosterone levels back in check first (which would have been if you had done a proper PCT). Correct me if I am mistaken, this is just what I’m gathering based on the information you have provided. Essentially, when you finished your Ostarine cycle, you had elevated estrogen and lowered test, you didn’t do anything about it and forgo’d any PCT, then before you had recovered properly you started another cycle with an already out of whack hormone profile, further compounding your issue. The PCT product certainly is not what has caused your issue, that I can assure you.
“Synthetic oxytocin is sold as proprietary medication under the trade names Pitocin and Syntocinon, and as generic oxytocin. Oxytocin is destroyed in the gastrointestinal tract, so must be administered by injection or as nasal spray. It has a half-life of typically about three minutes in the blood, and given intravenously does not enter the brain in significant quantities – it is excluded from the brain by the blood–brain barrier. Evidence in rhesus macaques indicates oxytocin by nasal spray does enter the brain. Oxytocin nasal sprays have been used to stimulate breastfeeding, but the efficacy of this approach is doubtful.
Injected oxytocin analogues are used for labor induction and to support labor in case of difficult parturition. It has largely replaced ergometrine as the principal agent to increase uterine tone in acute postpartum hemorrhage. Oxytocin is also used in veterinary medicine to facilitate birth and to stimulate milk release. The tocolytic agent atosiban (Tractocile) acts as an antagonist of oxytocin receptors; this drug is registered in many countries to suppress premature labor between 24 and 33 weeks of gestation. It has fewer side effects than drugs previously used for this purpose (ritodrine, salbutamol, and terbutaline).
The trust-inducing property of oxytocin might help those who suffer from social anxieties and mood disorders, but with the potential for abuse with confidence tricks and military applications.”